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PANCE® is a registered trademark of the National Commission on Certification of Physician Assistants (NCCPA). This application is an independent study resource and is not affiliated with, sponsored by, or endorsed by the NCCPA.

For educational use only. Content is AI-generated, may contain errors, and is not medical advice — always verify against current primary sources and faculty guidance before clinical use.

HomeRenalAcute Kidney Injury
🫘

Acute Kidney Injury

Renal · ~5% of blueprint
🔬Pathophysiology
Abrupt ↓ GFR — prerenal (hypoperfusion), intrinsic (e.g., ATN), or postrenal (obstruction).
🩺Signs & Symptoms
Often detected only by rising creatinine; ↓ urine output may be the earliest sign; edema; uremia if severe.
🎯Gold Standard Diagnosis
Rising creatinine / ↓ urine output (KDIGO); urine studies & FENa to localize the cause.
💊First-line Treatment
Treat the cause; optimize volume/perfusion, stop nephrotoxins; dialysis for refractory complications.
High-Yield Pearl

FENa <1% suggests prerenal; >2% suggests ATN; dialyze for refractory K+, volume or uremia.

Sources

Reviewed September 2026
  1. KDIGO Clinical Practice Guideline for Acute Kidney Injury — KDIGO, 2012
  2. KDIGO AKI/AKD Clinical Practice Guideline - Public Review Draft (not authoritative) — KDIGO, 2026
  3. StatPearls: Acute Kidney Injury — StatPearls Publishing (NCBI Bookshelf), 2024

Checked against the guideline or standard reference the exam keys to. If it has moved, use Report an error below.

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